Oxymorphone Hydrochloride Addiction and Abuse
Oxymorphone hydrochloride achieves its effects by binding to two opioid receptors in the brain: the mu opioid receptor (MOR) and the delta opioid receptor (DOR). Oxymorphone hydrochloride is produced from thebaine, a minor constituent of the poppy plant (Papaver somniferum).
The extended-release version of oxymorphone hydrochloride is indicated for the treatment of chronic pain. It’s typically only prescribed to patients who are already taking less powerful painkillers. Individuals who take the extended-release version may also be administered the immediate-release version during flare-ups of severe pain.
Oxymorphone hydrochloride is extremely habit-forming. When the pleasurable effects of the drug recede, the patient’s brain may crave more of the drug. As use continues and tolerance increases, the patient will need to take higher and more frequent doses to achieve the same effects.
Oxymorphone hydrochloride triggers the release of dopamine when it enters the bloodstream. This sidesteps the brain’s natural pathways. Over time, these pathways begin to deactivate in favor of the external stimulation provided by oxymorphone hydrochloride. The patient’s brain ultimately stops producing dopamine without the presence of oxymorphone hydrochloride and it can be difficult for the patient to experience pleasure without the drug.
Other medications can be given to help mitigate specific withdrawal symptoms. Doctors may prescribe anti-nausea medications for gastrointestinal distress or sleeping pills for the treatment of insomnia. Medications may also be prescribed for the treatment of anxiety and depression that are associated with withdrawal.
If you have any questions regarding opioid abuse and detox, The Recovery Village is available to answer them. Contact our confidential helpline 24 hours a day at 352-771-2700.
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